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Other studies show that: QA produces excitotoxic, axon-sparing lesions in the striatum that selectively deplete GABA and substance P while sparing somatostatin/neuropeptide Y neurons [88]

68 In addition to GLS inhibitors, other factors related to glutamine metabolism are promising targets for cancer therapy

Such selective interference distinguishes S-nitrosylation from broader oxidative damage and may underlie the molecular basis of ER-linked neurodegeneration