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Altogether, these findings position adipose tissue browning as a putative mechanistic axis through which semaglutide may exert its pleiotropic actions in metabolic disease, warranting further validation in studies focusing on human adipose tissue
doi: 10.1038/377646a0 102 TennantMKThrasherJBTwomeyPABirnbaumRSPlymateSR
Further reciprocal mutagenesis studies will be required to identify key interactions responsible for the contrasting effects of exendin-phe1, exendin-asp3 and other analogs, facilitating future design of agonists with even greater selectivity
Ferrannini, E., Rosenbaum, M
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