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Research on COPD endotypes is advancing rapidly, and several specific endotypes have been proposed or identified, including the T2-low endotype (neutrophil-predominant), T2-high endotype (eosinophil-predominant), genetic endotypes (1-antitrypsin deficiency endotype, telomerase gene mutation endotype), pulmonary vascular endotype, and persistent inflammation/autoimmune endotype ( Phenotypes represent the external clinical manifestations of COPD, while endotypes provide mechanistic explanations behind these manifestations, thus linking clinical observations to molecular pathogenesis
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In uveal melanoma, it has been demonstrated that lactate supplementation to in vitro cultured cells increases the mRNA expression of factors involved in mitochondria biogenesis and activity, suggesting a metabolic shift towards an oxidative metabolism [32]
