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The incorporation methylmalonyl-CoA into fatty acid biosynthesis results in branched-chain fatty acids being produced that may severely alter the architecture of the normal membrane structure of nerve cells

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In recent years, as a histone deacetylases (HDACs) inhibitor, it has been approved for being an adjunct to cancer therapy ( The mechanism of hepatotoxicity caused by VPA has been reported in many studies ( via different mechanisms ( When an impairment in -FAO pathway of fatty acids occurs, it causes the accumulation of the corresponding acyl-CoAs, resulting in an alteration of the corresponding ACs in plasma

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