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Estrogen regulates GLP-1 receptor expression in peripheral tissues, potentially altering the immune effects of GLP-1RAs and amplifying infection risk in a sex-specific manner [33]

The role that NAC may have in restoring normal levels of the inflammatory response is the result of a close interrelation between oxidative stress and inflammation, in whose response several mediators contribute, such as IL-1, IL-6, IL-10, TNF-, and NF-B, which mediate the redox-dependent regulation of inflammatory cytokines [4]

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Unlike heart disease or diabetes, fatty liver stays silent for decades